Literature Sources
Literature Sources
Chromodomains
CHD1 interacts with SSRP1 and depends on both its chromodomain and its ATPase/helicase-like domain for proper association with chromatin.
DNA-binding and chromatin localization properties of CHD1.
Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences.
A mammalian DNA-binding protein that contains a chromodomain and an SNF2/SWI2-like helicase domain.
Characterization of the CHD family of proteins.
CHD1 associates with NCoR and histone deacetylase as well as with RNA splicing proteins.
ISWI and Bromodomains
WSTF-ISWI chromatin remodeling complex targets heterochromatic replication foci.
Family of chromatin remodeling factors related to Williams syndrome transcription factor.
HuCHRAC, a human ISWI chromatin remodelling complex contains hACF1 and two novel histone-fold proteins.
A novel family of bromodomain genes.
Roles of Brahma and Brahma/SWI2-related gene 1 in hypoxic induction of the erythropoietin gene.
WSTF-ISWI chromatin remodeling complex targets heterochromatic replication foci.
Cloning of human and bovine homologs of SNF2/SWI2: a global activator of transcription in yeast S. cerevisiae.
A tissue-specific, naturally occurring human SNF2L variant inactivates chromatin remodeling.
The transcriptional regulation role of BRD7 by binding to acetylated histone through bromodomain.
Swi/Snf2 factors
A single-nucleotide polymorphism of SMARCB1 in human breast cancers.
Largest subunits of the human SWI/SNF chromatin-remodeling complex promote transcriptional activation by steroid hormone receptors
The MLL fusion partner AF10 binds GAS41, a protein that interacts with the human SWI/SNF complex.
BRG1, a component of the SWI-SNF complex, is mutated in multiple human tumor cell lines.
Recruitment of the SWI-SNF chromatin remodeling complex as a mechanism of gene activation by the glucocorticoid receptor tau1 activation domain.
Aberrations of the hSNF5/INI1 gene are restricted to malignant rhabdoid tumors or atypical teratoid/rhabdoid tumors in pediatric solid tumors.
Structure-function analysis of integrase interactor 1/hSNF5L1 reveals differential properties of two repeat motifs present in the highly conserved region.
P16INK4a is required for hSNF5 chromatin remodeler-induced cellular senescence in malignant rhabdoid tumor cells.
Cyclin E associates with BAF155 and BRG1, components of the mammalian SWI-SNF complex, and alters the ability of BRG1 to induce growth arrest.
BAF60a mediates critical interactions between nuclear receptors and the BRG1 chromatin-remodeling complex for transactivation.
The chromatin-remodeling complex WINAC targets a nuclear receptor to promoters and is impaired in Williams syndrome.
Reconstitution of a core chromatin remodeling complex from SWI/SNF subunits.